Acne? Bad skin?

NoAcnin, for stimulating skin renewal

  • NoAcnin is for both men and women.
  • NoAcnin is a natural product.
  • NoAcnin comes in a capsule taken orally.
  • NoAcnin targets latent viruses. Controlling latent viruses helps maintain healthy levels of DHT and androgen receptor in the skin, which stimulates skin renewal.†
  • NoAcnin has no side effects.†
  • If it does not work for you, you can always return it for a full refund.
  • The NoAcnin formula was developed analyzing thousands of scientific studies using artificial intelligence (AI).
  • NoAcnin is sold by Lilac Corp, the company that sells the highly acclaimed Gene-Eden-VIR and Novirin.


What Is NoAcnin?

NoAcnin is a patented herbal treatment. The formula includes 100 mg of a quercetin extract, 150 mg of a green tea extract, 50 mg of a cinnamon extract, 25 mg of a licorice extract, 130 mg of a curcumin extract, and 100 mcg of selenium. NoAcnin was launched at the end of 2017.

What is acne?

Acne is a very common skin condition; so common that it affects around 80% of young adults.  Although it usually appears during puberty, it can persist into adulthood with harmful effects on self-esteem.   Acne is found on the face, neck, back, and shoulders. [1]  The milder form of acne consists of whiteheads and blackheads.  The more severe form causes intense inflammation, lasts longer, and can leave scars. [2]

What causes acne?

Acne, in men and women, is caused by an increase in production of two proteins: 5α-reductase (5α-R) and androgen receptor (AR). [3],[4]

The 5α-R protein converts testosterone, the common male-sex hormone, into dihydrotestosterone (DHT). [3],[5]  Since there is more of the 5α-R protein in acne, there is also more DHT. 

Both testosterone and DHT bind to the androgen receptor (AR), but DHT is much better at binding. [6]  Since there is more DHT, and since there is more AR, there is more DHT protein bound to AR.

When DHT binds to AR, the new complex made of these two proteins sends a signal to the hair follicle to produce oil called sebum.  The abnormal level of sebum in the hair follicles clogs the hair follicle, causing the formation of whiteheads and blackheads.  The clogged follicle traps bacteria called Propionibacterium acnes, or P. acnes, which multiplies into high numbers and turns into inflammation and acne. [7]

As you remember, the initial event that starts acne is the increase in production of the two proteins 5α-R and AR.  What is the cause of this increase?

Latent viruses.  Viruses can be in a latent or active state.  Most people harbor a latent viral infection with more than 95% infected with EBV, more than 70% infected with CMV, and more than 90% infected with HSV-1 in many nations. [8]  Viruses in their latent state are often dismissed as harmless.  However, Hanan Polansky shows in his book ‘Microcompetition with Foreign DNA and the Origin of Chronic Disease’ that latent viruses can cause disease. [9]

According to Dr. Hanan Polansky, the genes of common latent viruses compete with the human genes for specific factors found in the nucleus of the cell.  The viruses are often stronger and win this competition.  Since these factors are rare, the viruses reduce the availability of these factors to the human genes.  This causes abnormal production of proteins in the human host.  Dr. Polansky called the fight between viral genes and human genes microcompetition. [9]

In the case of acne, microcompetition between the viral genes and the genes of the two proteins 5α-R and AR causes an increase in the production of these two proteins. [9]  When the two proteins reach a high level, the hair follicle produces a lot of sebum, resulting in inflammation and acne. [5]

How does NoAcnin work?

NoAcnin targets the latent viruses in the skin.  Controlling latent viruses  helps the body maintain normal levels of 5α-R, DHT, and AR, which helps the skin rejuvenate.†


[1] Lizneva D, Gavrilova-Jordan L, Walker W, Azziz R. Androgen excess: investigations and management. Best Pract Res Clin Obstet Gynaecol. 2016;37:98-118.

[2] Nast A, Dreno B, Bettoli V et al. European evidence-based (S3) guidelines for the treatment of acne. J Eur Acad of Dermatol Venereol. 2012;26(Suppl 1):1-29.

[3] Seiffert K, Setlmann H, Fritsch M, Zouboulis CC. Inhibition of 5α-reductase activity in SZ95 sebocytes and HaCaT keratinocytes in vitro. Horm Metab Res. 2007; 39(2):141-148.

[4] Durusoy C, Alpsoy E, Elpek O, Karpuzoglu G. Androgen receptor levels in the sebaceous glands of papulopustular lesions from patients with Behçet’s disease and acne vulgaris: a controlled study. Adv Clin Path. 2002;6(2):87-93.

[5] Lai J, Chang P, Lai K, Chen L, Chang C. The role of androgen and androgen receptor in the skin-related disorders. Arch Dermatol Res. 2012;304(7):499-510.

[6] Elsaie ML. Hormonal treatment of acne vulgaris: an update. Clin Cosmet Investig Dermatol. 2016;9:241-248.

[7] Barbaric J, Abbott R, Posadzki P et al. Light therapies for acne. Cochrane Database Syst Rev. 2016;9.

[8] Polansky H, Javaherian A. 3-Econsystems: MicroRNAs, Receptors, and La tent Viruses: Some Insights Biology Can Gain from Economic Theory. Front Microbiol. 2016;7:369.

[9] Polansky H. Microcompetition with Foreign DNA and the Origin of Chronic Disease. Rochester, NY: CBCD Publishing. 2003.